The Pill Millions of Women Take Daily For Joint Pain Is Making Their Joints Deteriorate Faster – A Nobel Prize Points to What Actually Works

If you’ve been taking ibuprofen, diclofenac, or naproxen for joint pain, this is worth reading before your next dose.

Not because they don’t work, but because of what happens after you’ve been taking them for a while.

Dr. Sandra Holt, a rheumatologist with 22 years of clinical experience specialising in autoimmune and joint conditions, has watched this play out in her clinic more times than she can count.

“Patients come to me after years on NSAIDs wondering why they’re worse than when they started,” she says. “The answer is usually sitting right there in their prescription history.”

“It remains largely unknown because it’s simply not as lucrative as the pharmaceutical alternatives,” she adds.

The practice itself is thousands of years old and the science behind it is serious enough that institutions like Harvard have spent $10 million studying it. ¹

The Research On NSAIDs That Pharmaceutical Companies Would Rather You Didn’t Read

Most doctors reach for nonsteroidal anti-inflammatory drugs (NSAIDs) – ibuprofen, diclofenac, naproxen – as the default answer to joint pain.

They work in the short term but the long-term picture is considerably darker.

One study found that long-term NSAID users were twice as likely to experience worsening pain and disability, and three times more likely to eventually need a total knee replacement. ²

Meanwhile, every NSAID except aspirin raises the risk of heart attack, stroke, and heart failure and that risk increases the longer you take them. ³

In other words: the standard treatment numbs the symptom while creating a bunch of other problems.

A Nobel winning solution that’s been hiding in plain sight for years

In 2016, Japanese cell biologist Yoshinori Ohsumi won a Nobel prize for his research on autophagy – a process of cells literally renewing themselves.

This process is primarily activated by fasting, periods of non-eating that can last anywhere between 16 to 72 hours.

This cell rejuvenation process is what gives joints the ability to become better through the following mechanisms.

#1 It reduces body inflammation

Fasting reduces inflammation at the source – and for many, that makes all the difference. In clinical observations, 36% of patients improved enough to stop pain medication altogether. ^{4 5 6}

#2 It slows cartilage breakdown and helps rebuild it.

Fasting triggers autophagy-driven cell regeneration, which clears out damaged cartilage cells and makes room for healthier ones. ⁷

In other words, fasting activates your body’s own repair system

Fasting can switch off joint inflammation but there’s a catch most people miss

Despite all the amazing research, fasting does have one negative.

Done wrong, it can spike cortisol, making joint pain worse.

That’s why so many women try it, feel nothing, and conclude it doesn’t work for them.

To actually make fasting work it needs to be tailored to your specific circumstances.

To your body and lifestyle.

Dr. Holt has reviewed this specific protocol that has a 90% satisfaction rate among her patients

Dr. Holt was asked to review a novel fasting protocol built around personalisation.

She reviewed the science, ran it through her own patient base, and tested the results firsthand.

Now she recommends it directly from her practice.

“The results have been consistent,” she says.

“90% of patients who followed it reported excellent improvement in joint pain and mobility.

Most of them also lost weight which is a nice bonus.”

It only works if it’s built for you

Before you start, the protocol has you take a short assessment. It’s there for two reasons:

#1 It analyses your profile to build a fasting structure specific to your biology.

#2 Completing it unlocks a private reader discount only available through this link.

The assessment takes two minutes. The results last a lot longer than that.